By Shafiqur Rahman

This quantity of Progress in Molecular Biology and Translational technology focuses at the molecular foundation of drug addiction.

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Opposing patterns of signaling activation in dopamine D1 and D2 receptor-expressing striatal neurons in response to cocaine and haloperidol. J Neurosci. 2008;28(22):5671–5685. 109. Xu S, Kang UG. Cocaine induces ubiquitination of Egr-1 in the rat dorsal striatum. Neuroreport. 2014;25(17):1362–1367. 110. Jenab S, Festa ED, Nazarian A, et al. Cocaine induction of ERK proteins in dorsal striatum of Fischer rats. Brain Res Mol Brain Res. 2005;142(2):134–138. 111. Sun WL, Zhou L, Hazim R, Quinones-Jenab V, Jenab S.

134. Edwards S, Bachtell RK, Guzman D, Whisler KN, Self DW. Emergence of contextassociated GluR(1) and ERK phosphorylation in the nucleus accumbens core during withdrawal from cocaine self-administration. Addict Biol. 2011;16(3): 450–457. 135. Suzuki A, Josselyn SA, Frankland PW, Masushige S, Silva AJ, Kida S. Memory reconsolidation and extinction have distinct temporal and biochemical signatures. J Neurosci. 2004;24(20):4787–4795. 136. Reichelt AC, Lee JL. Memory reconsolidation in aversive and appetitive settings.

The projections of the ventral tegmental area and adjacent regions: a combined fluorescent retrograde tracer and immunofluorescence study in the rat. Brain Res Bull. 1982;9(1–6):321–353. 5. Schultz W. Predictive reward signal of dopamine neurons. J Neurophysiol. 1998;80(1): 1–27. 6. Wise RA. Dopamine, learning and motivation. Nat Rev Neurosci. 2004;5(6):483–494. 7. Canales JJ. Stimulant-induced adaptations in neostriatal matrix and striosome systems: transiting from instrumental responding to habitual behavior in drug addiction.

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